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HomepageNavigationContentSitemapSearchDepartment of Health Sciences and TechnologyEnergy Metabolism LaboratoryStudent portalAlumni associationLoginContactenKeyword or personDepartmentsArchitecture and Civil EngineeringD-ARCH: Architecture D-BAUG: Civil, Environmental and Geomatic EngineeringEngineering SciencesD-BSSE: Biosystems Science and EngineeringD-INFK: Computer ScienceD-ITET: Information Technology and Electrical Engineering D-MATL: Department of Materials D-MAVT: Mechanical and Process Engineering Natural Sciences and Mathematics D-BIOL: Biology D-CHAB: Chemistry and Applied Biosciences D-MATH: Mathematics D-PHYS: Physics System-oriented Natural Sciences D-ERDW: Earth Sciences D-HEST: Health Sciences and Technology D-USYS: Environmental Systems Science Management and Social Sciences D-MTEC: Management, Technology and Economics D-GESS: Humanities, Social and Political Sciences Navigation Area About Us Overview Group Head Deputy Group Head Administration Postdoctoral Researchers Technical Personnel Doctorate Students People A-Z Open Positions How to Find Us Education Overview Research & Publications Overview Key Publications Complete Publication List Social Media Overview ETH Zurich D-HEST Energy Metabolism Laboratory Main content Our favorite aging model, the nematode C. elegans (enlarged) Welcome to the Energy Metabolism Lab We are pursuing research on the biochemical and molecular basis of longevity regulation to provide novel therapeutic options to prevent and cure age-related diseases like obesity, diabetes, neurodegeneration and cancer. We are interested in genetic pathways and environmental factors that modulate longevity. Besides other topics, we are particularly focused on the role played by mitochondria in lifespan regulation. In the past and contrary to the widely reiterated Free Radical Theory of Aging, we have repeatedly shown that the health-promoting effects associated with low caloric intake, physical exercise, sirtuins, impaired insulin/IGF-1 signaling, and other lifespan-extending interventions may be due to increased formation of Reactive Oxygen Species (ROS) within the mitochondria, causing a vaccination-like adaptive response that culminates in increased stress resistance and extended longevity, a process a. k. a. mitochondrial hormesis or mitohormesis. Based on this and meanwhile, we characterize identify and aging-associated pathways that have been identified by a RNA expression screen of physiological aging in several evolutionary distinct species, including C. elegans, zebrafish, killifish, and mice. Additional Information We are pursuing research on the biochemical and molecular basis of longevity regulation to provide novel therapeutic options to prevent and cure age-related diseases like obesity, diabetes, neurodegeneration and cancer. Read more Sitemap Imprint Disclaimer Copyright © 2016 Eidgenössische Technische Hochschule Zürich Page URL: http://www.energymetab.ethz.ch/ 16.12.
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